In kiwifruit orchards worldwide, the bacterial plant pathogen Pseudomonas syringae pv. actinidiae biovar 3 (Psa3) causes devastating canker disease. The global impact of this disease, with symptoms including blossom necrosis, cane dieback, trunk/cane bleeding, cankerous growths, and necrotic leaf spots, is a cause for concern for growers. To address this challenge, scientists have turned to gene editing as a promising solution.

A recent study published in Plant, Cell & Environment revealed that a gene known as NbPTR1a builds resistance against Psa3 infection in kiwifruit. Using gene editing technologies, the researchers developed an approach to bolster kiwifruit's natural defenses against Psa3. They discovered that a specific gene in tobacco plants, named NbPTR1, plays a crucial role in recognizing a protein produced by Psa3. This interaction triggers an immune defense response in the plant, effectively protecting it against the canker disease.

Through precise genetic modifications, researchers successfully introduced the NbPTR1 gene from tobacco into susceptible kiwifruit varieties. After evaluating the impact of expressing this gene, the study found that the transgenic kiwifruit exhibited increased survival rates and enhanced resistance to Psa3, compared to the wild-type plants.

The use of gene editing technologies to enhance crop resilience against pathogens like Psa3 marks a significant advancement in agricultural innovation. Strengthening kiwifruit's natural defenses is a crucial step in ensuring global sustainable production, as it enables farmers to safeguard yields against disease outbreaks.

Read the journal article in Plant, Cell & Environment for more information.